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Laurel has been given multiple awards for both her medical and legal accomplishments, as well as writing multiple research and educational publications.

Macabee Annual Award in Legal Medicine

10th World Congress on Medical Law

Jerusalem, Israel

August, 1994

American Jurisprudence Award in Commercial Law

February, l994

Young Investigator’s Award in Shock Research

Shock Society Annual Award

June, 1985

Chicago Surgical Society Research Award

Annual Research Award

May, 1985

Research Published as Lead Article, ANNALS OF SURGERY, 1985 Dec; 202(6):771-6.

The role of C5 in septic lung injury.

Olson LM, Moss GS, Baukus O, Das Gupta TK.

Excerpt: One proposed mechanism for the pathogenesis of lung injury in septic animals is the stimulation by C5a of granulocytes to produce and release toxic oxygen radicals that damage cellular membranes in pulmonary capillaries. The authors have investigated the possible role of C5 in septic lung injury, utilizing C5-sufficient and C5-deficient twin mice strains. In this lethal sepsis model, mean survival time is increased in C5-deficient mice in comparison to the survival of their C5-sufficient twins. Morphometric results demonstrate a significant increase in intracapillary granulocrit and air-blood barrier thickness 24 hours after cecal ligation and puncture in C5-sufficient septic mice. Similarly, mean arterial pO2 is significantly decreased in the C5-sufficient animals. Intracapillary granulocrit, air-blood barrier thickness, and arterial pO2 are normal in the septic C5-deficient twins of these animals. These data support the hypothesis that C5 is involved in the pathogenesis of septic lung injury.

Phi Beta Kappa

June 1976

Mortar Board Honor Society

May 1976

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